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This affinity may cause Proviron to displace other weaker substrates for SHBG (such as testosterone), another mechanism in which the free hormone level may be increased. Proviron (Ascorbic acid) – in women Proviron may act by mimicking estrogen or by blocking its synthesis by inhibiting the action of the hormone receptor (GPR30). Proviron's effects on ovarian volume could cause it atrophy and possibly infertility. In vitro, progesterone is reduced in progesterone-dependent animals such as rats, and reduced progesterone levels in women have been observed. The progesterone-deficient testes of women (such as in the ovary of women suffering from oligomenorrhea) are more prone to atrophy. In some of these women, ovarian and reproductive development appears altered in both male and female mice. Proviron has anti-androgenic activity by increasing the growth of the testicular stem cells. These cells may also affect the reproductive system (e.g. estrogenic effects). A study in young and old mice indicated that Proviron has the effect of increasing cell proliferation in the ovaries but not affecting the fertility of these mice. [8] Proviron binds to androgen receptors and inhibits the androgen binding protein, ADP-ribose (Abraxane or Agr. Rho in humans) and is able to bind androgen receptor subtypes in the ovaries (e.g. aromatase). Proviron also reduces the levels of estradiol and testosterone in the ovaries (progesterone). These two hormones and progesterone (which has no receptor specificity) are both essential to menstrual rhythm. Proviron can also exert effects on other sex characteristics. These include androgen preference (progesterone preference), prostate growth and development (testosterone/adrenomolysis), body composition (fatty liver), and hair development. Proviron affects the sexual characteristics of women in that it affects female breast cancer, endometrial morphology, and ovarian tissue size. Proviron also affects sexual characteristics in men. Proviron has anti-androgenic activity. [9] Similar articles:
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